The infundibulum is a stalk-like structure that connects the pituitary gland to the hypothalamus. It contains blood vessels and nerve fibers that facilitate communication between these two structures.

The pituitary gland produces hormones that regulate the function of other endocrine glands, influencing various physiological processes throughout the body.

The adenohypophysis, also known as the anterior pituitary, is responsible for producing and releasing hormones that regulate various physiological processes in the body.

Growth hormone (GH), also known as somatotropin, is produced by the adenohypophysis and plays a crucial role in growth and development, particularly in children and adolescents.

Prolactin (PRL) is produced by the adenohypophysis and is essential for stimulating milk production in lactating women.

Adrenocorticotropic hormone (ACTH) is produced by the adenohypophysis and stimulates the production of cortisol by the adrenal glands, which is essential for stress response and energy metabolism.

Thyroid-stimulating hormone (TSH), also known as thyrotropin, is produced by the adenohypophysis and stimulates the production of thyroid hormones by the thyroid gland, which are essential for metabolism and energy production.

Growth hormone (GH) is not stored in the neurohypophysis. It is produced and stored in the adenohypophysis.

Antidiuretic hormone (ADH) is primarily responsible for conserving water in the body by reducing urine output and increasing water reabsorption in the kidneys.

Oxytocin has two primary functions: stimulating uterine contractions during labor to facilitate childbirth and promoting milk ejection during breastfeeding.

A deficiency of growth hormone (GH) in children can lead to dwarfism, characterized by short stature and impaired growth.

An excess of growth hormone (GH) in adults can lead to gigantism, characterized by excessive growth and height.

A deficiency of prolactin (PRL) can lead to infertility, particularly in women, due to impaired milk production and ovulation.

An excess of prolactin (PRL) can lead to galactorrhea, characterized by the production of milk in non-lactating women.

A deficiency of adrenocorticotropic hormone (ACTH) can lead to Addison's disease, characterized by impaired production of cortisol by the adrenal glands.