Many herbicides work by inhibiting the process of photosynthesis in plants, preventing them from producing food and energy.

Most insecticides target the nervous system of insects, disrupting their normal function and leading to paralysis or death.

Organochlorines, such as DDT, are persistent organic pollutants (POPs) that can accumulate in the environment and pose risks to wildlife and human health.

Glyphosate inhibits the enzyme EPSPS, which is involved in the biosynthesis of aromatic amino acids, leading to the disruption of protein synthesis and plant death.

Insects can develop resistance to insecticides by increasing the production of detoxification enzymes that break down and eliminate the toxic compounds.

Pesticide nontarget effects refer to the adverse impacts of pesticides on organisms other than the intended target pest.

All of the mentioned strategies contribute to reducing the environmental impact of pesticides.

Neonicotinoids target and block nicotinic acetylcholine receptors in insects, leading to disruption of nerve transmission and ultimately causing paralysis and death.

Imidacloprid is a systemic pesticide that is absorbed by plants and translocated throughout their tissues, providing protection against pests that feed on the plant.

Pesticide drift, leaching, and volatilization are all processes by which pesticides can move through the environment.

All of the mentioned strategies contribute to managing pesticide resistance in insects.

Fungicides often target and inhibit the germination of fungal spores, preventing the establishment of fungal infections.

2,4-D is a selective herbicide that targets broadleaf weeds while leaving grasses relatively unaffected.

Pesticide drift refers to the movement of pesticides through the air, potentially contaminating areas beyond the intended target area.

All of the mentioned strategies contribute to reducing the risk of pesticide exposure to humans and the environment.